Abatacept in B7-1-positive proteinuric kidney disease.

نویسندگان

  • Chih-Chuan Yu
  • Alessia Fornoni
  • Astrid Weins
  • Samy Hakroush
  • Dony Maiguel
  • Junichiro Sageshima
  • Linda Chen
  • Gaetano Ciancio
  • Mohd Hafeez Faridi
  • Daniel Behr
  • Kirk N Campbell
  • Jer-Ming Chang
  • Hung-Chun Chen
  • Jun Oh
  • Christian Faul
  • M Amin Arnaout
  • Paolo Fiorina
  • Vineet Gupta
  • Anna Greka
  • George W Burke
  • Peter Mundel
چکیده

Abatacept (cytotoxic T-lymphocyte-associated antigen 4-immunoglobulin fusion protein [CTLA-4-Ig]) is a costimulatory inhibitor that targets B7-1 (CD80). The present report describes five patients who had focal segmental glomerulosclerosis (FSGS) (four with recurrent FSGS after transplantation and one with primary FSGS) and proteinuria with B7-1 immunostaining of podocytes in kidney-biopsy specimens. Abatacept induced partial or complete remissions of proteinuria in these patients, suggesting that B7-1 may be a useful biomarker for the treatment of some glomerulopathies. Our data indicate that abatacept may stabilize β1-integrin activation in podocytes and reduce proteinuria in patients with B7-1-positive glomerular disease.

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عنوان ژورنال:
  • The New England journal of medicine

دوره 370 13  شماره 

صفحات  -

تاریخ انتشار 2013